Trait
Sensory ataxia
Incoordination of movement caused by a deficit in the sensory nervous system. Sensory ataxia can be distinguished from cerebellar ataxia by asking the patient to close his or her eyes. Persons with cerebellar ataxia show only a minimal worsening of symptoms, whereas persons with sensory ataxia show a marked worsening of symptoms.
Summary
- Also called
- Afferent ataxia, Ataxia, sensory, Spinal ataxia
- Body system
- Abnormality of the nervous system
Reference genomeGRCh38hg38
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Get my Genome Test17 genes linked to Sensory ataxia
Changes in these genes cause rare conditions in which Sensory ataxia is seen. Most people with this trait have none of those conditions, and a gene on this list says nothing about anyone's own genome.
| Gene | How often it is seen | Conditions |
|---|---|---|
| COX18cytochrome c oxidase assembly factor COX18 | Always | In 1 condition |
| DNAJC3DnaJ heat shock protein family (Hsp40) member C3 | Always | In 2 conditions |
| EGR2early growth response 2 | Always | In 1 condition |
| MPZmyelin protein zero | Always | In 2 conditions |
| PMP22peripheral myelin protein 22 | Always | In 2 conditions |
| PRXperiaxin | Always | In 2 conditions |
| RNF170ring finger protein 170 | Always | In 1 condition |
| AARS1alanyl-tRNA synthetase 1 | Often (30–79%) | In 1 condition |
| KPNA3karyopherin subunit alpha 3 | Often (30–79%) | In 1 condition |
| NAGLUN-acetyl-alpha-glucosaminidase | Often (30–79%) | In 1 condition |
| POLGDNA polymerase gamma, catalytic subunit | Sometimes (5–29%) | In 1 condition |
| SH3TC2SH3 domain and tetratricopeptide repeats 2 | Sometimes (5–29%) | In 1 condition |
| TWNKtwinkle mtDNA helicase | Sometimes (5–29%) | In 2 conditions |
| UCHL1ubiquitin C-terminal hydrolase L1 | Sometimes (5–29%) | In 1 condition |
| FLVCR1FLVCR choline and heme transporter 1 | Not recorded | In 1 condition |
| GPIglucose-6-phosphate isomerase | Not recorded | In 1 condition |
| PIEZO2piezo type mechanosensitive ion channel component 2 | Not recorded | In 1 condition |
How often is among people with one of the gene's conditions, the most often across them, as the Human Phenotype Ontology records it. These pages describe traits and the research behind them; they are not a diagnosis, and a doctor or genetic counsellor is the person to ask about symptoms.
Broader traits
Where this comes from
- The Human Phenotype Ontology, release 2026-09-01: the trait, its names, its definition and the genes linked to it. Find out more at hpo.jax.org.
- Data release 2026.10.16-c6eea6bf.
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