Trait
Postprandial hyperglycemia
An increased concentration of glucose in the blood following a meal.
Summary
- Body system
- Laboratory abnormality
Reference genomeGRCh38hg38
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Read the genes linked to Postprandial hyperglycemia, and the rest of your genome, with one test.
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Get my Genome Test9 genes linked to Postprandial hyperglycemia
Changes in these genes cause rare conditions in which Postprandial hyperglycemia is seen. Most people with this trait have none of those conditions, and a gene on this list says nothing about anyone's own genome.
| Gene | How often it is seen | Conditions |
|---|---|---|
| INSRinsulin receptor | Very often (80–99%) | In 4 conditions |
| CACNA1Scalcium voltage-gated channel subunit alpha1 S | Often (30–79%) | In 2 conditions |
| GABRA3gamma-aminobutyric acid type A receptor subunit alpha3 | Often (30–79%) | In 1 condition |
| GYS2glycogen synthase 2 | Often (30–79%) | In 2 conditions |
| KCNE3potassium voltage-gated channel subfamily E regulatory subunit 3 | Often (30–79%) | In 1 condition |
| KCNJ18potassium inwardly rectifying channel subfamily J member 18 | Often (30–79%) | In 1 condition |
| SCN4Asodium voltage-gated channel alpha subunit 4 | Often (30–79%) | In 1 condition |
| SHPKsedoheptulokinase | Often (30–79%) | In 1 condition |
| SLC2A2solute carrier family 2 member 2 | Often (30–79%) | In 2 conditions |
How often is among people with one of the gene's conditions, the most often across them, as the Human Phenotype Ontology records it. These pages describe traits and the research behind them; they are not a diagnosis, and a doctor or genetic counsellor is the person to ask about symptoms.
Broader traits
Where this comes from
- The Human Phenotype Ontology, release 2026-09-01: the trait, its names, its definition and the genes linked to it. Find out more at hpo.jax.org.
- Data release 2026.10.16-c6eea6bf.
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